臨床上，根尖病灶中的細菌可能會對鄰近的植體造成齒源性感染，演變成植體-牙髓合併病灶。即使沒有放射可透性的病變，也不代表根尖組織中不存在致病微生 物 。對於植體而言，周圍的失活牙都是潛在的感染來源 (不論是否接受過根管治療)，而且無法以 X 光片判讀來達到完全的術前預防。再者，即使是活牙，也有可 能在植牙過程中致使其失活，進而演變出根尖病灶反過來影響植體的存活。本病例是一位 61 歲的女性患者，植體植入一個月後出現 Sussman Type 1 植體 - 牙髓合併病 灶；內文涵蓋臨床治療步驟、牙根尖 X 光片的變化，最後做討論與總結。

Clinically, microorganism in an apical lesion could be a potentially infectious source that could contaminate the surface of a nearby implant. We refer to this type of lesion as an implant-endodontic combined lesion. When we evaluate the risk factors of implants, adjacent devital teeth could be sources of infection, which cannot be prevented by preoperative X-ray examination. Furthermore, even vital adjacent teeth could be devitalized due to the surgical procedure of dental implantation, potentially resulting in an endodontic apical lesion that may be a hazard to the implant.
In our case report, a 61-year-old female patient encountered a Sussman’s type I endodontic-implant combined lesion, one month after implant insertion. This article draws conclusions about the overall clinical evolution (treatment procedure and X-ray findings), and provides a cumulative survey of related research, as well as discussion.