非酮病性高血糖引起單側舞蹈症病人之同側豆狀核有高密度之病灶

Presentation of Hemiballism-hemichorea in Non-ketotic Hyperglycemia with Ipsilateral Striatal Hyperintensity on CT and MR Images

運動障礙性疾病常因對側基底核病兆所引起。本文報告一例老年女性因非酮病性高血糖引起之單側舞蹈症。病患之腦部電腦斷層掃瞄顯示同側豆狀核有高密度之病灶；磁振造影檢查顯示在同側豆狀核有異常訊號之改變：在T1影像為高的訊號及在T2影像則是低的訊號。後續追蹤病患的腦部電腦斷層掃瞄顯示，在四個月後高密度病灶完全消失。

Movement disorder is often caused by lesion in the contralateral basal ganglia. This paper reports an old woman with hemiballism-hemichorea induced by non-ketotic hyperglycemia and her neuroimaging studies presenting with an ipsilateral lesion. A brain computed tomography revealed a hyperdense lesion in the ipsilateral putamen, while magnetic resonance image study revealed an abnormal signal change in the ispilateral putamen; hyperintensity on Ti-weighted and hypointensity on T2-weighted images. She was treated with insulin to control her hyperglycemia, and fellow-up brain CT 4 months later showed complete resoluation of the abnormal attenuation, and no recurrent episodes of hyperkinesia was noted.